Chronic fatigue syndrome: these intestinal bacteria that cause fatigue and chronic pain

A study from Columbia University in New York has shown that chronic fatigue syndrome (CFS) could be associated with an imbalance in the intestinal flora, the microbiota. Researchers have found abnormal levels of specific gut bacteria in people with CFS.

Some of the participants with CFS also suffered from irritable bowel syndrome (IBS). Sleep disorders, fatigue, CFS is a complex disease that weakens the patient. Signs of this syndrome include extreme fatigue after exertion and muscle and joint pain. It is estimated that up to 90% of people with CFS also have IBS.

Intestinal bacteria typical of chronic fatigue

American scientists from Columbia University noticed that levels of certain intestinal bacterial species – Faecalibacterium, Roseburia, Dorea, Coprococcus, Clostridium, Ruminococcus and Coprobacillus – were strongly associated with CFS. Their relative abundance seems to be a predictive way to diagnose CFS. The presence of certain bacteria, or the absence of others, also seems to help identify IBS.

These bacteria, which act as biomarkers, could make it possible to know the intensity of symptoms such as fatigue and pain in the event of CFS. To reach these conclusions, the New York scientists recruited 50 patients and 50 healthy people (like control group) in clinics specializing in the treatment of CFS.

Chronic Fatigue Syndrome: when certain bacteria cut the link with the brain

People with CFS have a mix of gut bacteria and associated metabolic disorders that can influence the severity of their disease. According to Prof. Brent L. Williams who conducted this study: “Our analysis suggests that we may be able to subtype patients with CFS by analyzing the faecal microbiota. “. “Subtypes can provide clues to understanding differences in disease manifestation.

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Much like irritable bowel syndrome, CFS could involve a breakdown in two-way communication between the brain and gut mediated by bacteria, their metabolites, and the molecules they influence. He concludes: “By identifying the specific bacteria involved, we are one step closer to more precise diagnosis and targeted therapies. This study was published in the scientific journal Microbiome.


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